Session Learning Objectives:

1. Describe the classical protease vs antiprotease hypothesis in the pathophysiology of AATD emphysema. 

2. Describe Z AAT polymer proteotoxicity may contribute to AATD emphysema. 

3. Discuss why induced pluripotent stem cell-derived AT2s are a good model of studying AATD. 

 

Carly Merrit, presenter, has no relevant financial relationships to disclose.

 

 

 

Session date: 
07/13/2022 - 3:30pm to 4:00pm EDT
Location: 
Zoom
United States
See map: Google Maps
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  • 0.50 AMA PRA Category 1 Credit™

    Boston University Chobanian & Avedisian School of Medicine designates this Live activity for a maximum of 0.50 AMA PRA Category 1 Credit™. Physicians should claim only the credit commensurate with the extent of their participation in the activity.

  • 0.50 Participation

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